Biology • Year 12 • Module 8 • Lesson 17

Prevention — Genetic Engineering, Screening and Emerging Technologies

Lock in the key vocabulary, classification of genetic disorders, inheritance patterns and diagnostic methods covered in this lesson.

Build · Vocab & Classification

1. Label the inheritance-pattern diagram

The diagram below shows a simplified summary of the four main inheritance patterns for single-gene disorders. Write the missing labels A–H into the spaces provided. Each label comes from the lesson's Key Terms or from the inheritance-pattern grid in Card 3. 8 marks

Diagram coming soon
  1. A — Name of pattern (top-left quadrant): _______________________
  2. B — Probability an AR couple (both carriers) has an affected child: _______________________
  3. C — Name of pattern (top-right quadrant): _______________________
  4. D — Probability an AD parent (heterozygous) passes condition to offspring: _______________________
  5. E — Name of pattern (bottom-left quadrant): _______________________
  6. F — Which sex is primarily affected in X-linked recessive disorders and why (one word): _______________________
  7. G — Name of pattern (bottom-right quadrant): _______________________
  8. H — Which parent transmits mitochondrial mutations to ALL children: _______________________
Stuck? Revisit the inheritance-pattern grid in Card 3 of the lesson.

2. Term–definition match

Write the correct term next to each definition. Choose from: genetic disorder, chromosomal abnormality, non-disjunction, single-gene disorder, penetrance, carrier, karyotype, NIPT, gene therapy, de novo mutation. 10 marks

#DefinitionMatching term
2.1A disease caused by an abnormality in an individual's DNA — either in chromosome number/structure or in a single gene sequence.
2.2Failure of homologous chromosomes or sister chromatids to separate during meiosis, producing aneuploid gametes.
2.3A display of an individual's chromosomes arranged by size and banding pattern, used to detect aneuploidy and structural rearrangements.
2.4An individual carrying one copy of a recessive disease allele who does not show symptoms of the condition.
2.5Non-invasive prenatal testing — analyses cell-free fetal DNA in maternal blood from 10 weeks gestation. A screening, not diagnostic, test.
2.6The proportion of individuals with a given genotype who actually show the associated phenotype.
2.7A disease caused by a mutation in one specific gene that follows a Mendelian inheritance pattern.
2.8A disorder arising from the wrong number or structural rearrangement of chromosomes (e.g. trisomy, monosomy, translocation).
2.9Experimental treatment that introduces, alters or replaces a gene within a patient's cells to treat or prevent disease.
2.10A mutation arising spontaneously during gamete formation or early embryogenesis — not inherited from either parent.
Stuck? Revisit the Key Terms panel at the top of the lesson.

3. True or false — with correction

Circle T or F. If the statement is false, write the corrected version on the line provided. 10 marks (1 T/F + 1 correction where needed)

3.1 Down syndrome (trisomy 21) is most commonly caused by a dominant mutation in a gene on chromosome 21.    T  /  F

3.2 NIPT is a diagnostic test that definitively confirms whether a fetus has a chromosomal abnormality.    T  /  F

3.3 Carriers of autosomal recessive conditions such as cystic fibrosis are phenotypically normal because their one functional allele produces enough protein to compensate.    T  /  F

3.4 Somatic gene therapy editing corrects mutations in the patient's gametes, so the change is passed to the patient's children.    T  /  F

3.5 Huntington's disease shows anticipation — the CAG repeat tends to expand in successive generations, causing earlier onset and greater severity in offspring.    T  /  F

Stuck? Revisit Cards 2, 3, 4 and 5 of the lesson and the Misconceptions box.

4. Function recall

Answer each in 1–2 sentences using precise lesson terms. 8 marks (2 each)

4.1 What is the function of amniocentesis in prenatal genetic testing, and how does it differ from NIPT?

4.2 What is the function of the Guthrie card (newborn screening program) and which conditions does it screen for in Australia?

4.3 What is the role of viral vectors in gene therapy, and name one approved gene therapy that uses them?

4.4 What is the function of CRISPR-Cas9 in treating genetic disease, and for which conditions was it first approved in 2023?

Stuck? Revisit Cards 4 and 5 of the lesson.

5. Fill in the blanks — cystic fibrosis mechanism

Complete the passage using words from the word bank below. Each word is used once. 8 marks

Word bank: CFTR, chloride, delta-F508, misfold, osmosis, mucus, autosomal recessive, chromosome 7

Cystic fibrosis (CF) is a(n) __________________ disorder caused by mutations in the CFTR gene located on __________________. The most common mutation is __________________, which causes the CFTR protein to __________________ and be destroyed before it reaches the cell surface. CFTR normally functions as a __________________ ion channel in epithelial cell membranes. Without functional CFTR, chloride ions cannot exit the cell; water follows by __________________, resulting in abnormally thick __________________ in the lungs, pancreatic ducts and reproductive tract. This ultimately leads to chronic lung infections and pancreatic enzyme deficiency.

Stuck? Revisit Card 3 (Cystic Fibrosis mechanism) in the lesson.

6. Build a concept map — genetic disorder classification

Draw labelled arrows between the six terms below to show how they connect. Each arrow must carry a linking phrase. Aim for at least 6 labelled arrows. 6 marks

Supplied terms: non-disjunction · chromosomal abnormality · trisomy 21 · CFTR mutation · single-gene disorder · cystic fibrosis

non-disjunction
chromosomal abnormality
trisomy 21
CFTR mutation
single-gene disorder
cystic fibrosis
Hint chain: non-disjunction → causes → chromosomal abnormality → example → trisomy 21; CFTR mutation → causes → single-gene disorder → example → cystic fibrosis.
Answers — Do not peek before attempting

Q1 — Inheritance-pattern labels

A: Autosomal Recessive (AR). B: 25% (1 in 4). C: Autosomal Dominant (AD). D: 50% (1 in 2). E: X-linked Recessive (XLR). F: Males (they have only one X chromosome, so a single recessive allele is sufficient to cause the condition; females need two copies). G: Mitochondrial. H: Mother (mitochondria are inherited via the egg, not sperm).

Q2 — Term–definition matches

2.1 genetic disorder • 2.2 non-disjunction • 2.3 karyotype • 2.4 carrier • 2.5 NIPT • 2.6 penetrance • 2.7 single-gene disorder • 2.8 chromosomal abnormality • 2.9 gene therapy • 2.10 de novo mutation.

Q3 — True / false with correction

3.1 False. Correction: Down syndrome (trisomy 21) is most commonly caused by non-disjunction during meiosis producing a gamete with an extra chromosome 21; it is a chromosomal abnormality, not a single-gene dominant mutation.

3.2 False. Correction: NIPT is a screening test — it identifies individuals at higher risk but cannot definitively confirm the condition. A positive NIPT result requires follow-up with a diagnostic test such as amniocentesis or CVS.

3.3 True.

3.4 False. Correction: Somatic gene therapy edits non-reproductive body cells only; it does not affect the patient's gametes and therefore the change is not passed to children. Only germline editing (currently banned in most countries) would be heritable.

3.5 True.

Q4.1 — Function of amniocentesis vs NIPT

Amniocentesis is a diagnostic test: a needle is inserted into the amniotic fluid (at 15–20 weeks) to obtain fetal cells, which are then karyotyped or analysed by PCR/DNA sequencing to confirm or rule out a specific genetic condition. NIPT is a screening test — it analyses cell-free fetal DNA in maternal blood (from 10 weeks) to identify pregnancies at higher risk of chromosomal abnormalities, but cannot provide a definitive diagnosis and carries no procedural miscarriage risk.

Q4.2 — Function of the Guthrie card

The Guthrie card is used in Australia's universal newborn screening program — a dried blood spot is collected from the baby's heel at 48–72 hours after birth and screened for metabolic and endocrine disorders including phenylketonuria (PKU), cystic fibrosis, congenital hypothyroidism and galactosaemia. Early detection allows treatment (e.g. phenylalanine-restricted diet for PKU) before symptoms develop.

Q4.3 — Role of viral vectors in gene therapy

Viral vectors (modified adeno-associated viruses [AAV] or lentiviruses) are used to deliver a functional copy of a defective gene into a patient's cells. The virus is stripped of its disease-causing genes but retains its ability to enter cells and integrate or express DNA. Approved examples include Luxturna (AAV carrying the RPE65 gene for a form of inherited blindness) and Zolgensma (AAV carrying SMN1 for spinal muscular atrophy).

Q4.4 — Function of CRISPR-Cas9 and first approved conditions

CRISPR-Cas9 acts as molecular scissors guided by a short RNA sequence to cut DNA at a precise genomic location, allowing a gene to be disrupted or corrected. In 2023, Casgevy (exa-cel) — the first CRISPR therapy approved globally — was approved for sickle-cell disease and beta-thalassaemia. It works by reactivating fetal haemoglobin production in the patient's own blood stem cells.

Q5 — Cloze passage answers (in order)

autosomal recessivechromosome 7delta-F508misfoldchlorideosmosismucus

(Note: the blank for CFTR in the opening sentence is already provided; the remaining 7 blanks use the seven remaining words.)

Q6 — Sample concept map

Acceptable arrows include:

  • non-disjunctionproduceschromosomal abnormality
  • chromosomal abnormalityexample istrisomy 21
  • non-disjunctioncan result intrisomy 21
  • CFTR mutationcausessingle-gene disorder
  • CFTR mutationspecifically causescystic fibrosis
  • single-gene disorderexample iscystic fibrosis

Award 1 mark per biologically correct labelled arrow with causal direction maintained. A maximum of 6 marks.